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HPMR

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High Mobility Group Protein 17 OKDB#: 1113
 Symbols: HMG17 Species: human
 Synonyms: CHROMOSOMAL PROTEIN, NONHISTONE, HMG17| NONHISTONE CHROMOSOMAL PROTEIN HMG17|  Locus: 1p36.1 in Homo sapiens


For retrieval of Nucleotide and Amino Acid sequences please go to: OMIM Entrez Gene
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General Comment Chromosomal protein HMG14 and its close analog HMG17 bind to the inner side of the nucleosomal DNA, potentially altering the interaction between the DNA and the histone octamer. The 2 proteins may be involved in the process that maintains transcribable genes in a unique chromatin conformation. Their ubiquitous distribution, relative abundance, and high evolutionary conservation of the DNA-binding domain of the HMG-14 family of proteins, suggest that they may be involved in an important cellular function. The human HMG14 multigene family is 1 of the largest retropseudogene families known.

NCBI Summary: Member of the HMG 14/17 family of proteins; may bind DNA with low specificity; shares a common DNA-binding motif with other HMG 1/2 family members
General function Cell death/survival, DNA Replication, Nucleic acid binding, DNA binding, Transcription factor
Comment
Cellular localization Nuclear
Comment
Ovarian function Oogenesis
Comment Mohamed OA, et al 2001 reported that High-mobility group proteins 14 and 17 maintain the timing of early embryonic development in the mouse. The high-mobility group (HMG) proteins 14 and 17 are abundant chromosomal proteins that bind to nucleosomes and enhance transcription. The authors reported that both mRNA species and both proteins are present throughout oogenesis and preimplantation development of the mouse.. The results identify HMG-14 and HMG-17 as constitutive components of mouse oocyte and embryonic chromatin and establish a link between the structure of embryonic chromatin and the normal progression of embryonic development.
Expression regulated by
Comment
Ovarian localization Oocyte
Comment
Follicle stages Primordial, Primary, Secondary, Antral, Preovulatory
Comment
Phenotypes
Mutations 0 mutations
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Phenotypes and GWAS show phenotypes and GWAS
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created: Feb. 22, 2001, 1:04 p.m. by: hsueh   email:
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last update: Aug. 13, 2001, 3:35 p.m. by: hsueh    email:



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