Stanford Home
Ovarian Kaleidoscope Database (OKdb)

Home

History

Transgenic Mouse Models

INFORGRAPHICS

Search
Submit
Update
Chroms
Browse
Admin

Hsueh lab

HPMR

Visits
since 01/2001:
176557

interleukin 16 OKDB#: 2830
 Symbols: IL16 Species: human
 Synonyms: LCF, NIL16, PRIL16, prIL-16,VWD|VON WILLEBRAND FACTOR DEFICIENCY|VON WILLEBRAND FACTOR, INCLUDED, VWF, INCLUDED|FACTOR VIII-VON WILLEBRAND FACTOR, INCLUDED, F8VWF, INCLUDED  Locus: 15q26.3 in Homo sapiens


For retrieval of Nucleotide and Amino Acid sequences please go to: OMIM Entrez Gene
Mammalian Reproductive Genetics   Endometrium Database Resource   Orthologous Genes   UCSC Genome Browser   GEO Profiles new!   Amazonia (transcriptome data) new!

R-L INTERACTIONS   MGI

DNA Microarrays
SHOW DATA ...
link to BioGPS
General Comment NCBI Summary: The protein encoded by this gene is a pleiotropic cytokine that functions as a chemoattractant, a modulator of T cell activation, and an inhibitor of HIV replication. The signaling process of this cytokine is mediated by CD4. The product of this gene undergoes proteolytic processing, which is found to yield two functional proteins. The cytokine function is exclusively attributed to the secreted C-terminal peptide, while the N-terminal product may play a role in cell cycle control. Caspase 3 is reported to be involved in the proteolytic processing of this protein. Alternate splicing results in multiple transcript variants. [provided by RefSeq, Feb 2010]
General function Ligand, Cytokine
Comment
Cellular localization Secreted
Comment
Ovarian function Initiation of primordial follicle growth
Comment Cytokine (IL16) and Tyrphostin Actions on Ovarian Primordial Follicle Development. Feeney A 2014 et al. An ovarian follicle is composed of an oocyte and surrounding theca and granulosa cells. Oocytes are stored in an arrested state within primordial follicles until they are signaled to re-initiate development by undergoing primordial to primary follicle transition. Previous gene bionetwork analyses of primordial follicle development identified a number of critical cytokine signaling pathways and genes potentially involved in the process. In the current study, candidate regulatory genes and pathways from the gene network analyses were tested for their effects on the formation of primordial follicles (follicle assembly) and on primordial follicle transition using whole ovary organ culture experiments. Observations indicate that the tyrphostin inhibitor BCI increased follicle assembly significantly, supporting a role for the mitogen activated protein kinase (MAPK) signaling pathway in follicle assembly. The cytokine interleukin 16 (IL16) promotes primordial to primary follicle transition as compared to the controls, where as Delta-like 4 (DLL4) and WNT-3A treatment have no effect. Immunohistochemical experiments demonstrated for both the cytokine IL16 and its receptor CD4 localization to the granulosa cells surrounding each oocyte within the ovarian follicle. The tyrphostin LDN193189 (LDN) is an inhibitor of the bone morphogenic protein receptor 1 (BMPR1) within the TGFB signaling pathway and was found to promote the primordial to primary follicle transition. Observations support the importance of cytokines (i.e. IL16) and cytokine signaling pathways in regulating early follicle development. Insights into regulatory factors affecting early primordial follicle development are provided that may associate with ovarian disease and translate to improved therapy in the future. /////////////////////////
Expression regulated by
Comment
Ovarian localization Granulosa
Comment
Follicle stages Primordial
Comment
Phenotypes
Mutations 0 mutations
Genomic Region show genomic region
Phenotypes and GWAS show phenotypes and GWAS
Links
OMIM (Online Mendelian Inheritance in Man: an excellent source of general gene description and genetic information.)
OMIM \ Animal Model
KEGG Pathways
Recent Publications
None
Search for Antibody


created: April 27, 2005, 6:35 a.m. by: hsueh   email:
home page:
last update: July 1, 2014, 11:04 a.m. by: hsueh    email:



Use the back button of your browser to return to the Gene List.

Click here to return to gene search form